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As medical doctors we often witness medical catastrophes. But rarely are we able to document the moment when the “eye of the hurricane” strikes. Read on and watch an echo you will probably never see again in your career.

Bad weather ahead

Manoj was suffering from chest pain for 4 days until he finally decided to come to our hospital. Typical ECG findings and positive cardiac enzymes made it clear: Manoj has a STEMI of the infero-lateral. The next step coronary angiography:


Not surprisingly Manoj had a tight lesion in the circumflex artery. All other coronary arteries were normal. Revascularization of circumflex artery was performed immediately:




The vessel is open with TIMI 2 flow after revacularisation


Manoj was stable and asymptomatic. So far so good – but unfortunately it was not the end of the story. On the 3rd post procedure day Manoj complained of abdominal distension and vomiting. His jugular pressure were elevated but the ECG did not show any significant changes. We immediately shifted him to the CCU and performed an urgent echocardiogram.





Blood, Blood, Blood

This is what we found as we put the transducer on the chest: The pericardium was filled with fresh blood.




Blood within the pericardial sack


Our first suspicion: cardiac tamponade. Manoj was deteriorating quickly. But let us look at the heart more closely. Here is a four chamber view that focus on the lateral wall of the left ventricle:






Dissected lateral wall and tamponade (pericardial effusion)


An effusion (blood) is seen lateral to the heart and the right ventricle is compressed - tamponade indeed. The septum and the apex are hyperdynamic. But look at the lateral wall: It is dyskinetic and dissected. Are we actually witnessing the moment of myocardial rupture?





The moment of rupture

I believe so. Look at the color Doppler study and you will see a jet from the left ventricle into the pericardial sack. I do not know of any study that has ever documented this before.




Color Doppler study with jet flow into the dissection and the pericardial sack


Manoj was immediately shifted to cardiothoracic OT. But, before undertaking any intervention he developed cardiac arrest, Despite CPR Manoj died within 20 minutes.





Cardiac rupture revisited

Cardiac rupture is a rare complication of acute myocardial infarction with an overall incidence of about 6.2%. Most patients with cardiac rupture may succumb almost instantaneously due to cardiac tamponade with rapid, irreversible, electromechanical dissociation. This is also the reason we rarely are able to document rupture. Only in small defects and if rupture is contained (as in the setting of a pseudoaneurysms) are we able to visualize rupture and even save patients.
In the present case the cause of cardiac rupture was thought to be related to a small branch of the left circumflex artery, which was occluded during PCI. This is one of the rare but important mechanisms of cardiac tamponade after PCI. It should also remind us that rupture more often occurs in small vs. large infarcts. What are some of the other findings associated with myocardial rupture?



  • Prolonged episode of anginal pain, often lasting longer than 6 hours
  • Shorter duration of pain but that has been preceded by other episodes longer than 30-60 minutes
  • Delayed hospitalization often because of a misdiagnosis of their AMI
  • Presence of maintained arterial hypertension (=>150 mm Hg) during the first 10-24 hours of the AMI
  • Performance of undue physical efforts, such as persistent coughing, vomiting or agitation

Despite the fact that you most likely will never see such an example again, it does highlight the fact that echo is the primary imaging technique in the setting of cardiac emergencies.
If you or your friends have similar rare cases, we are more than happy to post here. Let us use the collective wisdom, the power of the global community and the internet to teach, enlighten and make a difference to our patients…





Dr. Aniruddha De
Consultant Dept. of Non-Invasive Cardiology
Apollo Gleneagles Hospital
Dr. Thomas Binder
Department of Cardiology
Univ. of Vienna